الادارة فی أسواق القاهرة العثمانیة 1517-1579
نویسندگان
چکیده
منابع مشابه
Dmm019968 1579..1589
Oxidative stress in cardiac fibroblasts (CFs) promotes transformation to myofibroblasts and collagen synthesis leading to myocardial fibrosis, a precursor to heart failure (HF). NADPH oxidase 4 (Nox4) is a major source of cardiac reactive oxygen species (ROS); however, mechanisms of Nox4 regulation are unclear. β-arrestins are scaffold proteins that signal in G-protein-dependent and -independen...
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Non-progressive cerebellar ataxias are a rare group of disorders that comprise approximately 10% of static infantile encephalopathies. We report the identification of mutations in PMPCA in 17 patients from four families affected with cerebellar ataxia, including the large Lebanese family previously described with autosomal recessive cerebellar ataxia and short stature of Norman type and localiz...
متن کاملBio013276 1509..1517
Pseudohypoaldosteronism type II (PHAII) is a hereditary hypertensive disease caused by mutations in four different genes: with-no-lysine kinases (WNK) 1 and 4, Kelch-like family member 3 (KLHL3), and cullin 3 (Cul3). Cul3 and KLHL3 form an E3 ligase complex that ubiquitinates and reduces the expression level of WNK4. PHAIIcausing mutations in WNK4 and KLHL3 impair WNK4 ubiquitination. However, ...
متن کاملOp-brai130361 1579..1613
1 Queensland Brain Institute, The University of Queensland, Brisbane, 4072, Australia 2 Departments of Neurology and Pediatrics, The University of California and the Benioff Children’s Hospital, CA, 94158, USA 3 Departments of Pediatrics and Neurosurgery, Radiology and Biomedical Imaging, The University of California Children’s Hospital, CA 94143, USA 4 Departments of Paediatrics and Neurosurge...
متن کاملDmm021972 1517..1529
Tuberous sclerosis complex (TSC) is an autosomal dominant syndrome that is best characterised by neurodevelopmental deficits and the presence of benign tumours (called hamartomas) in affected organs. This multi-organ disorder results from inactivating point mutations in either the TSC1 or the TSC2 genes and consequent activation of the canonical mammalian target of rapamycin complex 1 signallin...
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ژورنال
عنوان ژورنال: مصر الحدیثة
سال: 2015
ISSN: 2314-8098
DOI: 10.21608/nmisr.2015.143249